Fat chance! Obese immigrants make America sicker.



It was one of those perfect Donald Trump social media posts — the kind that seems to straddle the line between truth and fiction, to bend and warp reality and make you ask, “Did he really just hit send on that?"

"Many in the fake news media have claimed that we will begin denying visas to overweight people," it began, before clarifying:

They have even come up with a term for these people, “High Calorie Humans.” This is TOTALLY FALSE. We will not ban all fat people from entering our Great Country, only those whose poor health will overburden our health care system. Visa applicants who are only slightly overweight have nothing to worry about. The bigger ones will need to trim down to get approved. We will EXPAND this rule to cover Expats in the near future.

The cherry on top was a closing swipe at one of Trump's favorite celebrity targets, currently in self-imposed exile in the Republic of Ireland: “Rosie, you will never return to This Great Country."

The US isn’t the first country in the world to limit entry to fat people. Other weight-watchers include Canada, Australia, and ultra-liberal New Zealand.

The phrase "High Calorie Humans" achieved instant "covfefe" status, as fans and haters alike reacted to the latest Trump provocation.

Fat shame

Except it was fake — a meme created in response to the very real news that the State Department has added obesity to the list of conditions that could bar foreigners from living in or visiting the U.S.

You know you're in the country's head when your constituents do your trolling for you. And you know you've hit a nerve when you dare suggest Americans could lose a few pounds — a form of truth-telling otherwise known as "fat-shaming."

But the U.S. is a country in which a Centers for Disease Control survey carried out between 2021 and 2023 found that a staggering 40.3% of adults were obese, with 9.4% having “severe” obesity.

That's using the standard metric of BMI, which uses height and weight to estimate body fat. But researchers have proposed including other anthropometric measurements in addition to BMI — such as waist circumference, waist-to-height ratio, and waist-to-hip ratio — for a more accurate assessment of obesity. Using this metric, the proportion of obese Americans could skyrocket to as much as 68.6%.

Heavy burden

No matter how you measure obesity, its direct medical costs are estimated to be some $170 billion a year, a figure that rises to more than $1.4 trillion when you consider the added effects.

And that's the burden Trump's new directive hopes to ease. With millions of overweight Americans already straining the country's health care system — and hitting taxpayers where it hurts — the last thing the country needs is to take on more patients from other countries.

America already rejects visa applications for those with conditions (like diabetes) that could make them a "public charge" — that is, someone likely to become dependent on government assistance.

The new directive builds on previous “public charge” rules, but it’s the first time obesity has been named specifically.

The directive applies primarily to immigrant visas — visas that will lead to long-term or even permanent settlement in the U.S. — and not non-immigrant visas (such as H-1B visas) or short-term tourist visas. Fat foreigners will still be able to visit the U.S. and work there. They just won’t be able to settle, unless they lose weight.

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Weight-watchers

The U.S. isn’t the first country in the world to limit entry to fat people. Other weight-watchers include Canada, Australia, and ultra-liberal New Zealand.

In one well-publicized case from 2009, the Kiwis denied residency to a British nurse who tipped the scales at 134 kg and had a BMI of over 50. A BMI of 25 is considered healthy. Officials estimated that her lifetime costs to the taxpayer could exceed NZ $800,000 or about US $500,000 at the time.

Canada and Australia have similar rules in place, but they receive much less attention.

Otherwise, though, there aren’t many “anti-fat” laws in effect worldwide. There’s Japan’s “Metabo” law, which came into effect in 2008. It is often described, misleadingly, as some kind of “ban” on fat people per se, but it’s not.

Instead, the law imposes an obligation on companies to ensure that workers between ages 40 and 74 receive an annual waistline measurement and help losing weight if they need it. Companies that don’t comply can be fined, but overweight workers themselves are not subject to any form of official punishment. In any case, Japan still has a remarkably low obesity rate, of around 4%.

Open borders for hotties?

I and other posters in my little corner of X have long called for restrictions of various kinds on overweight people, including proposals to prevent fat people from entering the country, in the name of beauty and the general welfare.

One of these proposals was even given the name “open borders for hotties”: If you’re fit and attractive (ideally female), you’re welcome, but if not — no thanks!

Critics will moan that Trump’s new rules for “HIGH-CALORIE HUMANS” are unfair and discriminatory, but frankly I can’t think of a policy that’s more in line with the fundamental MAGA principles. Immigration should benefit the nation, not sap its strength and resources. If a massively overweight person comes to the U.S. and the taxpayer has to fork out hundreds of thousands of dollars to cover his medical bills, what’s America First about that?

Instead of that fat person, a slim person with discipline and self-control could be brought into the country — or better yet, no immigrant at all. The money would be better spent elsewhere, and there are too many people in the country as it is.

The “HIGH-CALORIE HUMANS” rules are a clear sign, for all their apparent absurdity, that President Trump still understands what MAGA is and what it should stand for. Let’s see that understanding applied to immigration policy across the board and most of all to the H-1B visa system, which has been used for decades to disenfranchise and dispossess native workers. High calories, low wages — same difference.

'You know what really p**ses people off?' Vance identifies what's at heart of 'populist resentment' in Appalachia



Vice President JD Vance joined Health and Human Services Secretary Robert F. Kennedy Jr. at the Make America Healthy Again summit on Wednesday in discussing the Trump administration's revolution against the unworkable state of affairs and orthodoxies that have left so many Americans sick, censored, poor, and behind.

After the duo discussed President Donald Trump's penchant for taking "a bulldozer to Overton windows," Kennedy raised the matter of the dire health and social conditions in Appalachia, noting that Vance's incredible success serves as a "tragic reminder of the lost potential of almost everybody else in Appalachia."

'Their loved ones are dying much sooner than everybody else.'

"It's got the worst health data of any region in the country — the highest cardiac disease, the highest obesity, the highest diabetes, the highest stroke rates — but also addiction, alcoholism, and suicide," said Kennedy.

Although dubbed a "golden child of Appalachia" by the HHS secretary, Vance emphasized his firsthand familiarity with the bleak conditions experienced by so many in the region, noting that he was hard-pressed to identify a single important male family figure who lived past the age of 70.

"You want to talk about, like, 'populism'? And you want to talk about people being pissed off? Well, yeah, people are pissed off when they don't have good jobs; and people are pissed off when things disappear and move overseas; and people are pissed off when they feel like, you know, other countries are being prioritized over the United States of America," said Vance. "All of that is part of the populist resentment of the past 20 or 30 years in American politics."

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Photo by Alex Wong/Getty Images

"But you know what really pisses people off?" continued the vice president. "When they realize that their loved ones are dying much sooner than everybody else."

Life expectancy has long been lower and infant mortality higher in Appalachia than in the rest of the country.

Vance noted that while on the one hand, he feels guilty that so many of his fellow Appalachians have not enjoyed the opportunities for economic and familial stability that he has enjoyed, he also feels "a great sense of anger because we never should have gotten to the point that we are today, and the reason that we have is because of failed leadership — and it's failed leadership over generations."

The vice president stressed that one of the reasons he strongly supports Kennedy's health initiatives is because therein lies a major opportunity to do right by Appalachian residents who have been "left behind by this country's leadership."

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Can leucovorin cure autism? Meet the moms determined to find out



A humble, decades-old folate compound — used not to fight cancer but to ease the side effects of chemotherapy — has become the latest flashpoint in America’s health wars.

On September 10, the Trump administration announced that the FDA would move toward approving leucovorin for children with cerebral folate deficiency, a rare metabolic disorder linked to autism in some cases. Supporters hailed it as long-overdue recognition of promising small studies; critics called it another example of the MAHA agenda politicizing science.

While bureaucrats and scientists bicker, families with real skin in the game tirelessly run their own experiments and share their results, hoping the science will eventually catch up.

The debate since has been fierce, with professional groups such as the American Academy of Pediatrics advising against the off-label use of leucovorin for autism, warning that the evidence remains preliminary — while prominent physicians call for larger, biomarker-guided trials to confirm what early studies suggest.

A parent’s love

All parties insist their motives are pure, but this latest skirmish is a reminder of how tangled those motives can be. What drives the people and institutions pushing medical science forward is often a sincere desire to help people, yes — mixed in with ambition, rivalry, financial interest, and the unspoken urge to be the one who’s right.

But there’s another force at work here, deeper and simpler, and it tends to override all the rest: a parent’s love for a child.

This is the same love that kept the parents of children with cystic fibrosis pushing to understand a condition doctors considered hopeless, or that led a Hollywood father to resurrect a forgotten epilepsy therapy to help his son. And now it’s the force animating hundreds of parents who believe a decades-old folate compound has literally given their autistic children a voice.

While bureaucrats and scientists bicker, families with real skin in the game tirelessly run their own experiments and share their results, hoping the science will eventually catch up.

Even before the FDA signaled approval of leucovorin for cerebral folate deficiency — a rare metabolic disorder with links to autism — parents have been sharing reports of progress with the drug on Reddit forums and in Facebook groups to share anecdotal reports of progress. A few families have also told their stories in clinic-produced or news-segment videos.

A treatment’s hope

Leucovorin, also called folinic acid, is a bioactive form of folate. It’s been used for decades to “rescue” patients from high-dose chemotherapy. In autism, it’s being repurposed to bypass what some researchers call a “folate transport blockade.”

Up to 70% of autistic children in certain studies test positive for folate receptor alpha autoantibodies — immune proteins that prevent folate from reaching the brain. The result: cerebral folate deficiency. High-dose folinic acid appears to restore that supply, sometimes with striking behavioral effects.

Dr. Richard Frye, a pediatric neurologist at Phoenix Children’s Hospital, led one of the first controlled trials in 2016. His team found improved verbal communication in FRAA-positive children treated with leucovorin. Later case studies described language bursts, better eye contact, and calmer affect.

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Photo by ISSAM AHMED/AFP via Getty Images

From ‘no words’ to the Pledge of Allegiance

The parents themselves provide more affecting testimony. Carolyn Connor’s son Mason was 1 when she realized something was amiss: “He wasn’t talking. No language. No words.”

When their pediatrician downplayed this lag in development as typical in boys, she and her husband began doing their own research, which led them to Frye. Three days after starting leucovorin, Mason spoke his first words.

Now 6, he continues to take the medication, and continues to thrive.

Beth Ann Kersse’s daughter was diagnosed with autism at age 3. “In her vocabulary she had about three or four words,” Kersse said in a video uploaded by Washington, D.C.-based Potomac Psychiatry.

“But she didn’t call me ‘Mom.’ She kind of would point at me,” she added.

That’s when Kersse and her husband began exploring leucovorin. Two years later, Kersse describes her almost 5-year-old daughter’s transformation as “incredible.”

“The other day she stood up and put her hand over her heart, and she recited the Pledge of Allegiance, and we were just like, OK ... I didn’t know we knew that. ... She’s able to have a full conversation; she can tell us how she’s feeling.”

Late last month, Nebraska pediatrician Dr. Phil Boucher posted a case study detailing how a 3.5-year-old autistic girl responded to leucovin treatment, citing texts from her mother reporting that she was “blown away” by the changes she observed:

She is starting to consistently look at people when they call her name. ... She’s becoming more interested in her little sister. ... She also has started taking some of the baby dolls that we have and has been covering them up with a blanket, giving them a kiss, and saying, “Night night.”

As Boucher is careful to point out, anecdotal success stories like these don’t prove the drug works. But to those experiencing the improvement firsthand, they’re a promising sign that a simple, inexpensive vitamin derivative can do what years of therapy can’t.

And if this promise does indeed bear fruit, leucovorin treatment will be the latest of many homegrown revolutions in medical care spearheaded by determined mothers and fathers unwilling to wait for consensus.

It's the testosterone, stupid!



It was with great interest that I read Matthew Gasda’s latest essay, on the state of men in 2025, “Masculinity at the End of History.”

Gasda has a lot of things to say that are germane to my new book, "The Last Men: Liberalism and the Death of Masculinity" (out December 16), not least of all whether America — and indeed the Western world as a whole — exhibits what could be called a “crisis of masculinity" in the first place.

We have reams of data showing what can only be described as a civilizational decline in testosterone levels, a decline that may have no parallel in history.

There are plenty of observers — writers, social scientists, journalists, politicians, celebrity psychologists — who think so.

A crisis in need of a crisis

Gasda disagrees. In fact, he believes the absence of a crisis is precisely what’s ailing America’s young men. Men need crises in order to be men. Without crises, their mettle isn’t tested, they have no higher aspirations to direct themselves toward, and so they fall into a listless state, an aimless state, a kind of suspended adolescence.

Porn. Pot. Video games. Social media. Processed food. Logging on and dropping out. We all know what it looks like.

“Masculinity is desperate for a crisis,” Gasda writes in the opening paragraphs of his essay.

It is docile, unsure, and formless. At most, it is at the germinal phase of crisis, lacking a catalytic agent to propel it to its full-blown state, which at least can be registered and reckoned with. After all, crisis implies that something is happening, that something is at stake. The uncatalyzed proto-crisis, or the noncrisis, of American masculinity is repressed, unexpressed, yet omnipresent.

It’s a typical literary switcheroo — Gasda is a playwright, after all — but he’s not wrong. Nor is he the first to say that what men really need is a crisis — read: something extraordinary — to give full form to their potential.

Declaring 'war'

Back in 1910, the pragmatist philosopher William James, brother of the novelist Henry, wrote an essay called “The Moral Equivalent of War.” A committed socialist and pacifist, James nevertheless regretted the march of progress and with it the (apparent) decline of war, because he recognized war’s power to form young men and inculcate in them the highest possible virtues. War teaches men to subordinate themselves and their needs to those of the collective, to pursue a higher goal, and, if need be, to give their lives for it. War teaches men courage, service, self-sacrifice, stoicism, and patriotism, and all of these things are necessary for a properly functioning nation in peace.

But war is also a terrible, terrible thing — and it was rapidly becoming much worse, though just how much worse James could not have foreseen. What we need, James argues, is a “moral equivalent” of war, a substitute that could teach men the same lessons without the enormous destructive cost.

James’ proposal is quite clever: Rather than a war against each other, we need a war with nature. Young men should be enlisted into a national struggle to conquer and tame nature and to revolutionize the means of production. Send boys off to build railroads and skyscrapers and ships, and they’ll return as men, ready to lead families and the nation.

Manufacturing manhood

This isn’t too different, actually, from what Gasda advocates in his new essay, when he says a national project in which all or many men could participate might be a great spur to masculine revival.

If the objective of America in the years ahead is to reclaim global leadership in industrial production, that is, in the making of things in the real-world economy, as opposed to just in the realm of bits and pixels, then new avenues for masculine exertion, discipline, creativity, and camaraderie may arise from such a project.

There’s much to like in Gasda’s essay and much to agree with. He’s right about how the breakdown of communities and the loss of tradition have hindered the transmission of masculine ideals across the generations. He’s right about the need for rites of passage to confer status on men. Countless anthropological studies have shown the crucial role, in virtually every kind of society except our own, of tests of courage and fortitude at key moments in life, and psychologists have demonstrated how pain and trauma bond people together and provide a sense of shared identity.

He’s also right to argue that Americans must “historicize” masculinity. That is, they must understand its peculiar focus on strenuous exertion and relentless self-making in its particular historical context: a masculine ideal developed in conflict with a frontier, both the physical frontier of western expansion and the social and moral frontiers of a new national identity.

And he’s right, obviously, that we live in an age that’s fundamentally hostile to expressions of masculinity and that we can’t simply return to the past and past ideals, as so many simple-minded critics of the modern world, especially on social media, seem to believe.

That’s all to the good. But there are also serious problems.

No country for men

For one thing, it’s not clear just how much American men really could get behind a drive to, in Gasda's words, “reclaim global leadership in industrial production.”

If America does return to industrial pre-eminence, most if not nearly all manufacturing is going to be high-tech and automated — hardly the kind of gigantic Soviet five-year plan that could simply swallow up millions of men and give them jobs in factories or even give them jobs at all.

It's not just manufacturing that is on the verge of making human labor largely a thing of the past. Whole swaths of industry and even white-collar fields are undergoing the same revolutionary changes. Librarians and lawyers and proofreaders and doctors will be replaced by AI and large language models too.

The testosterone decline

A far graver problem, from my perspective, is that like the vast majority of the so-called “crisis of masculinity” literature that he derides, Gasda fails to take seriously, or even acknowledge, the biological changes that are throwing men’s masculinity into doubt — in particular, a headlong decline in testosterone, the master male hormone that’s responsible for making men men and not women.

Testosterone is not just responsible for sexual differentiation — for the physical characteristics that define boys, beginning in the womb and proceeding through infancy and the teenage years into adulthood — but it also governs male mood, motivation, libido, and even things like political attitudes.

Although we should be careful not to say testosterone determines political views, social psychology experiments reveal that a testosterone boost will make a man more likely to defend his position even when he’s outnumbered by people who disagree with him; it will make him more likely to continue fighting against a much stronger opponent; it will make him more accepting of hierarchy and inequality; it will make him more generous to his in-group — his own people — and more aggressive toward his out-group — potential enemies.

In short, testosterone and its effects are complex, but they work in ways that obviously tend toward behavior we associate with traditional masculinity. The less of it men have, the less masculine they become, as a basic rule.

Aggressively overlooked

Open a best-selling book like Richard Reeves’ "Of Boys and Men," head to the index, and look for “testosterone,” and you’ll find a poverty of references. Reeves talks about testosterone for just a few pages, but only to dispel the notion that boys “are their hormones,” meaning boys aren’t doomed to be aggressive because they have more testosterone (pop science’s “aggression hormone”) than girls. That’s it. Apparently, biology just isn’t important when we’re talking about the serious problems with men today.

It’s a strange oversight. We have reams of data showing what can only be described as a civilizational decline in testosterone levels, a decline that may have no parallel in history. We know what this decline entails, and if we don’t, we really should try to find out.

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Compelling evidence

The first real herald of a civilizational decline in testosterone levels was the Massachusetts Male Aging Study, a gold-standard double-blind controlled study of men in the Boston area. The study took place over a period of around 20 years, from the end of the 1980s to the early 2000s. Men of all ages were selected at random and given a battery of tests at regular intervals. When the testosterone data was finally analyzed in 2007, it showed testosterone levels were declining year over year at a rate of about 1%.

That might not sound like much, but over a period of 20 years, that’s 20%, or one-fifth. On a longer timeline, say 50 years, that’s half of all testosterone — gone.

Researchers in other countries, including Finland and Israel, wanted to see whether the same trend was happening in their countries. In Finland, where male reproductive parameters are generally better than in the U.S., the researchers believed the Boston trend would not be replicated. Guess what? The trend was actually worse, and the researchers showed it was taking place over a much longer period of time. The results of the MMAS were replicated in Israel, too, and in other American studies.

Quantifying maleness

It’s hard to quantify exactly how many men have low testosterone, in large part because nobody agrees on exactly how little testosterone counts as low. Ask one doctor and he’ll give you one figure; another will tell you it’s half or double that amount.

Symptomology is generally the best way to go looking for low testosterone, and what we see, everywhere we turn, is men who look and behave like they have low testosterone.

In Japan today, there are millions of hikikomori, or extreme social recluses — young men who simply refuse to participate in society. They hide themselves away at home, often with their parents, and play video games, eat junk food, and just “rot,” to use a current term.

At least one expert believes there may be as many as 10 million hikikomori, in a nation of 120 million people — that’s one in 12 people. Unsurprisingly — to me at least — research has shown young Japanese men are at significantly greater risk of becoming hikikomori if they have low testosterone.

America has its hikikomori too, although they aren’t called that. Maybe as many as 6 million, by some estimates.

Some of them congregate in special subforums on the website Reddit, like r/lowT, where they discuss what it’s like to be a man with low testosterone: how they have no motivation, no libido, can’t sleep, can’t get an erection, are developing gynecomastia — man boobs — and are overweight and anxious all the time.

Many of these men also describe the miraculous effects of increasing their testosterone, more often than not through a doctor’s prescription of testosterone in gel or injectable form.

Spermageddon?

What’s even more worrying about this decline is that it’s part and parcel of a broader decline in reproductive health parameters among men.

This isn’t a surprise: If men’s testes aren’t functioning properly and producing enough testosterone, they’re unlikely to be producing enough of other important things either. Sperm counts and sperm quality — a measure of sperm’s ability to swim properly and do their job — are declining so rapidly that one expert, Professor Shanna Swan, is predicting a “spermageddon” scenario, in which humans are unable to reproduce by natural means.

Swan made this the subject of a 2021 book, "Count Down." Simply by extrapolating the data for sperm-count decline, Swan has shown that by around 2050, the median man will have a sperm count of zero. One half of all men will produce no sperm at all, and the rest will produce so few that they might as well produce none, because they won’t be able to get a woman pregnant, try as they might.

What’s causing these changes? It’s lots of different things, a whole range of lifestyle factors — lack of exercise, smoking, bad diets, poor sleep, stress — but also widespread exposure to harmful chemicals known as “endocrine disruptors,” for their negative effects on the body’s hormonal (endocrine) system.

From low-T to trans

When I say endocrine disruptors are everywhere, I mean it: They’re in the food, the air, the water, the clothes we wear, our bedding and furniture, the deodorants and fragrances we put on our bodies, the little scented trees we put in our cars, anything that’s made from plastic.

A significant proportion of these harmful chemicals directly or indirectly mimic the effects of the hormone estrogen, interfering with the body’s crucial hormonal balance (more testosterone and less estrogen for men, the opposite for women). This is a nightmare for both sexes. As well as reducing testosterone and fertility in men, exposure to endocrine disruptors can lead to genital abnormalities, weight gain, and metabolic issues and even certain kinds of cancer.

New research has linked exposure to endocrine disruptors during gestation to transgenderism. French boys exposed to the chemical diethylstilbestrol, which used to be given to mothers at risk of miscarriage, had a massively increased risk — perhaps as much as a hundredfold — of undergoing gender transition later in life. On paper, it was always plausible that exposure to endocrine disruptors should be linked to gender dysphoria, but since transgenderism is such a toxic issue politically, there’s been little desire, until now, to pursue research into the link.

In a very real sense, then, not only have we created a society where masculinity is ridiculed, dragged through the mud, and denounced as retrograde, we’ve also created one where the biological constituents of masculinity, its very building blocks, are under direct attack at the same time. It’s a complicated problem, and it’s viciously circular. Biology and society exist in feedback loops, with negative effects reinforcing each other, deepening the spiraling decline.

While Gasda, like William James before him, may be right that men need a crisis to bring out the best in them, the very real danger today is that when one finally comes, men won’t have the energy or enthusiasm or desire to put down the controller, stand up, and answer its call. And if that really is the case, testosterone — the lack of it — will be to blame.

SSRIs and mass shootings: A link we can't afford to dismiss



There’s no link between antidepressant use and mass shootings, at least not according to a new study published in the journal Psychiatry Research.

Certainly good news for the pharmaceutical industry — but does one study really mean case closed?

The FDA’s own adverse event reporting system shows a consistent link between SSRIs and violence among adults.

It’s a controversial topic that has only become more so in recent years, especially now that Robert F. Kennedy Jr. is the secretary of health and human services under President Trump.

Deplorable questions?

Kennedy has long maintained that antidepressants are causing mass shootings. In an interview with Elon Musk in 2023, for example, Kennedy said, “Prior to the introduction of Prozac [a selective serotonin reuptake inhibitor], we had none of these events [mass shootings].”

In his confirmation hearings in February, he told the Senate that the link “should be studied, along with other possible culprits.”

He was asked to clarify his views about antidepressants and mass shootings by his Democrat interrogators, because they were intended to be discrediting just by being uttered aloud — just like his views on water fluoridation, vaccination, and the origins of COVID-19. A whole basket of deplorable ideas.

In short, we’re talking about yet another partisan political issue, even though, surely, we can all agree that mass shootings are terrible and we need to do everything we can to stop them.

Guns, not drugs?

And that includes, obviously, understanding what motivates the shooters.

The new study looked at over 800 mass-shooting incidents that took place between 1990 and 2023. The researchers used publicly available data — news reports, court records, and police statements — to see whether the perpetrators had any history of antidepressant or psychotropic drug use and whether there was a link between suicidality and mass shootings. Previous research had suggested there was such a link.

The researchers found evidence of lifetime antidepressant use in just 34 out of 852 cases and evidence of psychotropic drug use more broadly in 56 cases — just 6.6%. There was no unusual association between suicidality and mass shootings either. Suicide attempts were slightly more common among those with a history of medication use, but the difference was not statistically meaningful.

Population-level data also indicated that antidepressant use among mass shooters was lower than among the general public. If antidepressants were causing mass shootings, we’d expect levels of antidepressant use to be higher, not lower.

QED — or so the researchers believe.

“The vast majority of mass shootings have nothing to do with mental illness,” Ragy R. Girgis, one of the study authors, told medical news website PsyPost.

“The primary modifiable population-level risk factor for mass shootings is firearm availability.”

Prevent people from getting their hands on guns, prevent mass shootings. It’s that simple.

Or is it?

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Premature conclusions

There’s a glaring problem: The data simply isn't good enough to allow any kind of firm conclusion to be reached. The writers at "PsyPost" do at least acknowledge there’s a serious problem, although it doesn’t stop them from trumpeting “new study finds no evidence” in their headline.

Here’s what "PsyPost" says about the reliability of the evidence on offer.

Data were collected from publicly available sources, such as news articles and online records. This approach may miss cases where medication use was not reported or was kept confidential. The study also could not determine whether medications were being taken as prescribed during the attack or whether the person had recently stopped taking them.

Data is often kept confidential, even in the most high-profile cases. Take the Columbine shooters, Eric Harris and Dylan Klebold. At the time of the massacre, which claimed the lives of 14 people and left another 20 wounded, it was widely reported that Harris had been on the powerful new SSRI Luvox, generic name fluvoxamine. The New York Times claimed Harris had been rejected by Marine recruiters just five days before the attack for taking the drug.

There were suggestions that he had tried to go cold turkey as a result and that this might have affected his actions on that dreadful, bloody day. The Times noted that “patients taking Luvox are warned that if combined with other drugs, including alcohol, the drug can cause extreme agitation progressing to delirium, coma and death. The package also carries a warning about suicide.”

While officials said neither shooter had drugs or alcohol in his system at time of death, the coroner refused to say whether they had been tested for antidepressants, including Luvox.

And so we still don’t know, 26 years later, whether antidepressants played a role in the Columbine killings.

Mandatory screenings

Thankfully, there are now some attempts to provide answers. Unsurprisingly, they’re coming from Republican politicians and red states.

Tennessee has become the first state in the U.S. to introduce mandatory screening for psychotropic drugs in mass killings, defined as incidents in which four or more people are killed. In every mass killing that takes place in Tennessee, a detailed toxicology report will be produced and made available to the public. Investigators will study drug interactions in the killer’s body — because drugs have different effects when used in combination, a fact that is poorly understood — and they’ll also consult with providers of mental health services if the killer was receiving treatment.

Here’s something we do know for sure. A clear, well-established link exists between SSRIs and all forms of violent behavior. A huge Swedish study from 2020 that looked at 250,000 people revealed a significant association between SSRI use and violent crime, especially among 15- to 24-year-olds and 25- to 35-year-olds. The study also showed that risk of violence remained elevated up to 12 weeks after discontinuation of the drugs. The FDA’s own adverse event reporting system shows a consistent link between SSRIs and violence among adults.

A tall order

Instead of dismissing the possibility of a link between antidepressant use and mass shootings, we actually need to do some proper research. Gather data and interpret it objectively — meaning dispassionately, without imposing an ideological agenda that fixes the conclusions in advance.

I know that’s a tall order, given how emotional a subject mass murder is — especially mass murder of children — and how unwilling we all are to talk across the growing political divide, but that’s the scientific ideal, and that’s the only way we’re ever going to get to the truth.

As every first-year history undergraduate knows — and I was one, once upon a time — absence of evidence is not evidence of absence. Let’s not get twisted. Lives are at stake.

Hello, darkness, my old friend: How to get your body's circadian rhythms back on the beat



A few weeks ago, I enjoined all of you men to do what the right-wing bodybuilders and broscientists of X do and sun your scrota, treating the testicles within to certain health-giving frequencies of light.

As silly as that might sound, red-light therapy is a pretty hot trend, and exposing your genitalia to it probably has testosterone-boosting effects. This week, I’m going to be counseling you avoid light — and not just for your nether regions.

A study from last year found a 'significant relationship between outdoor light pollution and Alzheimer’s disease prevalence.'

The fact is that we’re exposed to too much light, of the wrong kind and at the wrong times, and it’s seriously screwing with our bodies and minds.

Apeman

My attitude to health, in a nutshell, would be this: Try to live, as much as you can, in the manner of your ancestors. Why? Well, because we're the same as them, more or less. We Homo sapiens haven't changed much from our days as hunter-gatherers 200,000 years ago. We’re still running more or less the same firmware, with a few important updates here and there.

What is different, however, is the environment you inhabit. It really kicked off about 10,000-12,000 years ago, with the Agricultural Revolution in the Near East. Now there were these things called farms and cities, and there was commerce and administration and taxes, armies and wars.

Once agriculture started to spread, the pace of change really started to pick up. Fast-forward to about 200 years ago and the dawn of the Industrial Revolution, and it starts to get really crazy.

All of a sudden you’ve got people living in communities of millions and a mind-boggling complexity. You’ve got people working in factories, eating food made in factories, wearing synthetic clothing made in factories, illuminating their homes and communities with electric light 24/7, bombarding their bodies with electromagnetic radiation 24/7, dosing themselves with ever-increasing numbers of medications, inhaling and swallowing billions of tiny pieces of plastic invisible to the naked eye.

The modern age

I’m not going to list all the changes here. The truth is that our bodies simply can’t cope — not fully — with the vast transformation our way of life has undergone in recent centuries and especially in the last hundred years. Keep in mind that this "modern" way of life is but a blip in human time. Our genes haven’t caught up yet, and maybe they never will. So it pays to treat your body, as much as you can, in the manner of your ancestors — to eat like them, move like them, and do the things they did.

Of course, you can go too far in your pursuit of an ancestral lifestyle — just look at the sad, sad story of the Liver King, a true cautionary tale for the “primal” community.

And I’m not saying the modern world doesn’t have its beguiling or useful aspects. I rather like the fact that I’m here, in my centrally heated living room, writing this article on my swish little iPad with its touch screen and magnetic keyboard. That’s pretty cool and useful. Then again, I suppose I could get used to writing in the manner of, say, Cicero: reclining on a couch in a toga, dictating to a slave while another one feeds me grapes.

Who loves the sun

The changes that have taken place in terms of light in the last century or so — to how we use and relate to light, natural and artificial — have been no less drastic than the changes to any other aspect of our lives, including the way we eat.

For the vast majority of human history, we organized our lives consciously and unconsciously around the natural diurnal rhythms of the planet. We woke up with the sun, and we went to bed when it became dark. There were seasonal changes, but they repeated, year after year. The invention of artificial light — fire, candles — didn’t really affect any of that much.

It was only with electric light that it become possible to defy the natural rhythms of night and day and the seasons and become what we are today: beings in possession of perpetual suns we can use to illuminate ourselves and our surroundings as much as we want, whenever we want.

Night moves

Banishing the darkness has had dramatic effects on our health.

Light governs the body’s circadian rhythms, also known as the “body clock,” which play a key role in regulating the secretion of hormones and processes of growth and recovery.

The main frequencies of light emitted by screens and LED lighting are blue, and these seem to have particularly bad effects on our bodies. Some scientists have suggested that blue light should be considered an endocrine disruptor — something as prone to mess with our hormones as nasty plasticizing chemicals, herbicides and pesticides, and many of the additives we find in ultra-processed food.

A study in the journal Environmental Research associates blue-light exposure with increased rates of breast cancer among women who do night work and sleep disorders among teenagers.

RELATED: LED astray: Yes, those harsh lights are the spawn of Satan

Photo by Bloomberg/Getty Images

Blue arrangements

In my piece on the boon of ball bronzing, I mentioned a study showing that chronic exposure to blue light could actually bring on early puberty in rats. This is a pretty worrying finding, especially since we know the age of puberty in the developed world has been decreasing for decades and children are being exposed to ever-greater quantities of blue light from the screens and electronic devices they play with all day long.

Large-scale studies clearly suggest exposure to artificial light could be having population-level effects. Research has linked nighttime light exposure to cognitive decline, for example. A study from last year compared rates of Alzheimer’s to satellite data for nighttime light levels across the U.S. The scientists found a “significant relationship between outdoor light pollution and Alzheimer’s disease prevalence. States and counties with higher levels of artificial light at night consistently had higher rates of Alzheimer’s disease.” Most worryingly of all, the association was strongest in the under-65s, a demographic that typically doesn’t suffer from this terrible disease.

So what can you do?

Doctor my eyes

In general terms, you should try to reduce your exposure to artificial light and do things that mimic the natural rise and fall of light levels. Go out and get sunlight early in the morning, or expose yourself to bright light with a SAD lamp. As afternoon draws into evening, begin to reduce levels of light in your home or workspace. Tell your body it’s getting closer to sleepy time. Turn off the main lights, turn on lamps, close the curtains — you could even light a few candles. I light my kitchen in the evening with candles, and it creates a wonderfully relaxing atmosphere. Begin to wind down your use of electronic devices that emit blue light.

If you have to spend your day or large portions of it staring at a screen, you can buy a pair of blue-light-blocking glasses. Ra Optics makes some very fetching blue-light blockers that don’t look at all silly. In fact, they’re basically indistinguishable from normal glasses or sunglasses. You could also buy one of Daylight’s very swanky tablets that has a blue-light-free backlight.

Alternatively, you can play with your device’s brightness settings or download a blue-light app like f.lux or Twilight. There’s also a built-in feature on Windows called “Night Light” that allows you to reduce levels of blue light in the evenings. You can even toggle it to come on automatically at set times.

Of course, you could just turn the bloody thing off. Imagine that. Our ancestors, fortunate creatures that they were, didn’t have to.

Trump administration claims link between autism and Tylenol, greenlights remedy



President Donald Trump noted in his order establishing the Make America Healthy Again Commission that "autism spectrum disorder now affects 1 in 36 children in the United States — a staggering increase from rates of 1 to 4 out of 10,000 children identified with the condition during the 1980s."

The MAHA Commission's chairman, Health and Human Services Secretary Robert F. Kennedy Jr., subsequently indicated that Centers for Disease Control and Prevention data show that the rate of autism among American children has gotten even worse.

"The autism epidemic is running rampant," Kennedy said. "One in 31 American children born in 2014 are disabled by autism. That's up significantly from two years earlier and nearly five times higher than when the CDC first started running autism surveys in children born in 1992. Prevalence for boys is an astounding 1 in 20, and in California it's 1 in 12.5."

'So taking Tylenol is not good.'

Kennedy promised during a Cabinet meeting in April that "by September, we will know what has caused the autism epidemic, and we'll be able to eliminate those exposures" — an ambition the Autism Society of America said was "harmful, misleading, and unrealistic."

Despite pre-emptive criticism by medical establishmentarians and the protest of the interim CEO of Tylenol maker Kenvue, Kennedy joined President Donald Trump and Centers for Medicare and Medicaid Services Administrator Mehmet Oz on Monday in formally identifying one of the alleged drivers behind the rise in American autism: the use of acetaminophen, the active ingredient in Tylenol, during pregnancy.

Kennedy, who indicated that his agency is also looking closely at the potential link between vaccines and autism, noted, "The FDA is responding to clinical and laboratory studies that suggest a potential association between acetaminophen use during pregnancy and adverse neurological outcomes, including later diagnoses for ADHD and autism."

RELATED: Trump's health revolution: RFK Jr. takes aim at chemicals, junk food, and overmedication

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The health secretary indicated that the U.S. Food and Drug Administration kicked off the process on Monday for a label change for acetaminophen to indicate that the use of the drug by pregnant women may be associated with an increased risk of neurological conditions like autism and ADHD in children.

The Department of Health and Human Services will also launch a nationwide public information campaign to alert parents and families to the possible risks of taking Tylenol during pregnancy, Kennedy said.

"The safety of acetaminophen against the risk of neurodevelopmental disorders in young children has never been validated," Kennedy said. "Prudent medicine therefore suggests caution in acetaminophen use by young children, especially since strong evidence also has associated it with liver toxicity. Some studies have also found the use of acetaminophen in children can potentially prolong viral illnesses."

"So taking Tylenol is not good," Trump said. "I'll say it: It's not good."

White House spokesman Kush Desai said in a statement to Blaze News, "President Trump pledged to address America's rising rate of autism, and to do so with gold-standard science. Today's announcement will make historic progress on both commitments."

Ahead of the announcement, a spokesperson for Kenvue — whose company stock price took a nosedive on Monday — told Blaze News, "We believe independent, sound science clearly shows that taking acetaminophen does not cause autism. We strongly disagree with any suggestion otherwise and are deeply concerned with the health risk this poses for expecting mothers."

The company spokesperson suggested further in the statement: "The facts are that over a decade of rigorous research, endorsed by leading medical professionals and global health regulators, confirms there is no credible evidence linking acetaminophen to autism."

Christopher Zahn, chief of clinical practice at the American College of Obstetricians and Gynecologists, similarly suggested in a statement to Blaze News prior to the Trump administration's announcement regarding autism that "there is no clear evidence that proves a direct relationship between the prudent use of acetaminophen during pregnancy and fetal developmental issues."

'Failure to implement change in medical practice currently constitutes disregard for the ample evidence of harm.'

While Kenvue, the ACOG, and other outfits have suggested that there is no causal link between acetaminophen use and autism, there is at the very least an apparent association.

In a National Institutes of Health-funded 2019 study published in the journal JAMA Psychiatry, researchers led by Dr. Xiaobin Wang of the Johns Hopkins Bloomberg School of Public Health collected umbilical cord blood from 996 births and measured the amount of acetaminophen and two of its byproducts in each sample.

The researchers found that "cord biomarkers of fetal exposure to acetaminophen were associated with significantly increased risk of childhood ADHD and ASD in a dose-response fashion."

Acetaminophen, often sold under the brand Tylenol in the United States and Canada, is the most common over-the-counter pain and fever medication used during pregnancy and is reportedly used by well over 50% of pregnant women worldwide.

A 2023 scientific review published in the Swiss peer-reviewed journal Children concluded "without reasonable doubt and with no evidence to the contrary that exposure of susceptible babies and children to acetaminophen (paracetamol) induces many, if not most, cases of autism spectrum disorder."

The review, led by Dr. William Parker, CEO of WPLab and visiting scholar at the University of North Carolina at Chapel Hill, also concluded that "the very early postpartum period poses the greatest risk for acetaminophen-induced ASD, and that nearly ubiquitous use of acetaminophen during early development could conceivably be responsible for the induction in the vast majority, perhaps 90% or more, of all cases of ASD."

When asked about Kenvue's apparent denial of a causal link between acetaminophen and autism, Dr. Parker told Blaze News:

Technically, the company is correct. Acetaminophen alone absolutely does NOT cause autism. Susceptibility to injury is absolutely required for acetaminophen to induce autism. Without susceptibility, which is caused by a very complex mixture of genetics, epigenetics, and environment, acetaminophen cannot induce autism. Almost all scientists working in the field are aware to some extent of the complex mix of genetic and environmental factors involved in the induction of autism.

In his response, Dr. Parker also referenced a 2024 study that he worked on which was published in the journal Clinical and Experimental Pediatrics. The study noted that "careful examination reveals no valid objections to the conclusion that early exposure to acetaminophen causes neurodevelopmental injury in susceptible babies and children."

"Changes in medical practice should be implemented that effectively weigh the risks and benefits of neonatal and pediatric APAP use," the study reads. "Failure to implement change in medical practice currently constitutes disregard for the ample evidence of harm despite the absence of any valid rationale for the view that APAP might be safe for neurodevelopment."

On the matter of whether health officials should warn pregnant mothers about the increased risk of autism in their children associated with acetaminophen use, Dr. Parker noted that the "answer to this question is nuanced."

"Evidence indicates that heavy use of acetaminophen during pregnancy may lead to neurodevelopmental problems, including autism and ADHD," Dr. Parker said. "Heavy use is often associated with chronic pain management. Much less is known about the cost-to-benefit ratio of treating an occasional fever during pregnancy. Such treatments may have a net benefit for the fetus, although more work needs to be done to probe this topic."

Dr. Parker emphasized to Blaze News that "we are absolutely NOT blaming parents and physicians for this. It is not a mistake for us to do what we are told is best for our children. The science is all about preventing injury in the future, not blaming people who are blameless."

In an NIH-supported study published in August in the peer-reviewed medical journal Environmental Health, researchers from the Harvard T.H. Chan School of Public Health, University of California Los Angeles' School of Public Health, and the Icahn School of Medicine at Mount Sinai systematically reviewed 46 "well-designed" studies incorporating data from over 100,000 participants regarding the relationship between neurodevelopmental disorders, including autism and attention-deficit/hyperactivity disorder, and prenatal exposure to acetaminophen.

The researchers found that 27 of the studies reported "significant links" between acetaminophen exposure in the womb and NDDs and noted that "higher-quality studies were more likely to show positive associations."

"Overall, the majority of the studies reported positive associations of prenatal acetaminophen use with ADHD, ASD, or NDDs in offspring, with risk-of-bias and strength-of-evidence ratings informing the overall synthesis," the study reads.

When specifically evaluating the studies pertaining to Tylenol use and autism in children, the researchers found "strong evidence of a relationship between prenatal acetaminophen use and increased risk of ASD in children."

Dr. Andrea Baccarelli, a co-author of the study and a professor of environmental health at the Harvard T.H. Chan School of Public Health, noted in a statement obtained by Blaze News that he believes "caution about acetaminophen use during pregnancy — especially heavy or prolonged use — is warranted."

Dr. Baccarelli and his colleagues recommended a "balanced approach" regarding acetaminophen use during pregnancy — a recommendation now echoed by the Trump administration: "Patients who need fever or pain reduction during pregnancy should take the lowest effective dose of acetaminophen, for the shortest possible duration, after consultation with their physician about their individual risk-benefit calculation."

Zahn of the ACOG was among those who railed against Baccarelli's systematic review.

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Photo by SAUL LOEB/AFP via Getty Images

"Failing to treat medical conditions that warrant the use of acetaminophen is, at present, understood to be far more dangerous than theoretical concerns based on inconclusive reviews of conflicting science," Zahn said in a statement to Blaze News. "Maternal fever, diagnosis of severe pre-eclampsia, and appropriate pain control are all managed with the therapeutic use of acetaminophen and can create severe morbidity and mortality for maternal and child health if they are mismanaged based on improper clinical recommendations."

The Society for Maternal-Fetal Medicine also rushed to endorse Tylenol use during pregnancy following the publication of the damning review, noting that it "continues to advise physicians and patients that acetaminophen is an appropriate medication to treat pain and fever during pregnancy."

"Ideally, you don't take it at all," Trump said during the press conference on Monday. "If you can't tough it out or there's a problem, you're going to end up doing it."

In addition to calling out acetaminophen for its alleged role in the explosion of autism cases, Kennedy identified leucovorin, which is also known as folinic acid, as a viable autism treatment.

Leucovorin is already used to treat cerebral folate deficiency, which has been associated with autism.

The same year that a review in the Journal of Personalized Medicine noted that leucovorin "is associated with improvements in core and associated symptoms of ASD and appears safe and generally well-tolerated," a double-blind, placebo-controlled trial was registered. The results of that trial, published last year in the European Journal of Pediatrics, were promising.

Researchers concluded that oral folinic acid supplementation "is effective and safe in improving ASD symptoms, with more pronounced benefits in children with high titers of folate receptor autoantibodies."

FDA Commissioner Dr. Marty Makary indicated that his agency has initiated the approval of leucovorin calcium tablets for patients with cerebral folate deficiency.

"We have witnessed a tragic four-fold increase in autism over two decades," Makary said. "Children are suffering and deserve access to potential treatments that have shown promise. We are using gold standard science and common sense to deliver for the American people."

The biopharmaceutical company GSK promptly noted that it will submit a supplemental New Drug Application for leucovorin to update the label to reflect that it can be used to treat cerebral folate deficiency.

By addressing one of the alleged root causes of autism and mainstreaming a treatment might not only help American families tackle the disorder but spare them from what is, for many, a crushing burden.

Upwards of $60.9 billion are reportedly spent each year on children with ASD, and intensive behavioral interventions can cost anywhere from $40,000 to $60,000 per child annually.

Blaze News has reached out to the American Pediatric Society and to the HHS for comment.

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Sweat equity: The surprising health benefits of a hot bath



On the rare occasions I can face visiting London these days, I always make sure to stop in at Banya No.1 in Hoxton. From the outside, it’s unprepossessing — just another modern block with a buzzer door — but when you get inside and go downstairs, it’s like you’ve been transported beyond the new Iron Curtain to Moscow or St. Petersburg or Novgorod.

It’s a proper Russian spa — a banya — complete with sauna, huge wooden-barrel plunge pool, freezing showers, treatment rooms with tables, and even a little cafe where you can sit and gobble pelmeni (delicious little stuffed dumplings) or have a hearty bowl of borscht (beetroot stew with meat).

Sauna treatment has been used, with great success, to treat police officers suffering chronic health issues as a result of repeated exposure to methamphetamine, for example.

And, of course, there are the staff, all of whom are authentic Russki, from the charming girls on the desk to the sullen myrmidons who patrol the saunas and abuse you with various implements for an extra fee — although you get the feeling they’d happily abuse you without payment too.

It’s a shame, but also a blessing, that I don’t live close to London. I’d be at Banya No. 1 all the time. If money were no object, I’d recreate it at home. I’d have a fragrant cedar-clad sauna with a big brick oven in it, and I’d pay a couple of former goons from the vory v zakone called Andrei and Pavel to keep the wood burning and beat me with birch twigs and scour me with Siberian pine tar to my heart’s content. Heaven.

I’d do this not just because it’s fun and a potent way to reduce stress — and we could all do with a bit less of that right now — but also because going to the spa is good for you in other ways.

Toxic baste

You’ve probably heard talk of the detoxification benefits of sauna before. They’re very real, and as I try to impress on my readers as much as I can, we all need to be detoxing our bodies. We’re constantly exposed to harmful chemicals in our daily lives, and anything we can do to reduce the amounts that get into and accumulate in our bodies is a good thing.

Sweating is one of the principal routes for harmful chemicals to leave our bodies, as well as in our urine and feces and when we lose (or give) blood. Particular chemicals, such as bisphenol A (BPA), a plasticizer with endocrine-disrupting effects, are generally found in greater quantities in our sweat than in our urine or blood, which is a clear sign that sweating is the main way our bodies dispose of them.

Sauna treatment has been used, with great success, to treat police officers suffering chronic health issues as a result of repeated exposure to methamphetamine, for example. It’s being trialed as a treatment for a range of other health conditions.

Fat chance

Sweating appears to be particularly effective for detoxification when combined with fasting or a calorie-restricted diet. Rodent studies show that being in a fasted or dieting state pushes harmful chemicals out of the body’s diminishing fat stores and toward the skin, where they can then be sweated off.

Many harmful chemicals are lipophilic, and as a result, when they get into our bodies, they end up in our fat stores. That’s actually one of the many important functions of fat — as a kind of vault for harmful chemicals, to seal them away and keep them out of other tissues — and it’s probably why a reliable effect of regular exposure to harmful chemicals is to put on fat. It’s a protection mechanism.

(This is also probably one of the reasons why celebrities who go on drugs like Ozempic end up looking so terrible, even when they lose loads of weight — search for before-and-after pictures of podcaster Ethan Klein if you don’t want to sleep tonight. Because of their terrible diets and lifestyles, fat people have enormous stores of toxic chemicals in their bodies, and if they suddenly lose weight with Ozempic or Mounjaro or whatever, their tissues are flooded with them. This could actually be quite dangerous, and it’s one of the many reasons you should prefer traditional methods of weight loss — exercise and changes to diet — over these new drugs when possible.)

Hot air

If you want to sauna regularly, you have various options. Of course, you can join a gym or health club with a sauna. That’s easy enough. If you want to sauna at home and you’ve got space in your garden, you can get a wooden barrel sauna for a few thousand dollars or convert a shed or outbuilding. You’ll find plenty of good guides online for building your own sauna.

If you don’t want to build a permanent structure, you could go the Native American route and build a little sweat lodge in your garden with a tent and some rocks. Dig a hole, put a tent (preferably canvas) over it, fill the tent with hot rocks heated on a fire, sit inside the tent, and pour water on the rocks. Voila: steam in a confined space. A sauna. Again, you’ll find helpful tutorials all over the internet.

There are also infrared sauna kits that can be used indoors and folded away and stored easily if you don’t have a great deal of space. They’re pretty reasonable.

But there may also be an even simpler and cheaper option. Just have a hot bath.

Tub-thumping

Yes: A hot bath probably confers all the benefits of a sauna. In fact, it may even be more effective at boosting your health than a sauna, according to a new study I wrote about recently on my Substack. It’s worth noting that the study didn’t compare the detoxification effects of baths and saunas due to sweating, but if you’ve ever sat in a hot bath, you’ll know you sweat plenty. Sweating takes place underwater, too, but you just don’t feel it.

What the new study does show is that hot baths offer superior metabolic, cardiac, and immune benefits to saunas, in large part because the heating effect of being immersed in hot water is much, much greater than sitting in a heated enclosure. Water conducts heat 24 times more efficiently than air, and your whole body — or most of it — is in direct contact with the water the entire time.

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Jason Connolly/Raymond Boyd/Getty Images

Save our swimmers

I have one caution about hot baths, though, and it’s for my male readers. Protect the family jewels. Boiling them is even worse than toasting them (see my note about testicular tanning in a previous installment).

Why? Heating the testicles is associated with reduced fertility, whether as a result of working in a furnace or as a welder, or through taking regular hot baths or saunas. The effects can be long-lasting, even after just a single session.

One study from the 1960s showed that it takes between 10 and 12 weeks for sperm counts to return to normal after just 30 minutes of intense scrotal heating. If you’re going to take regular hot baths as a man, I’d recommend keeping your testicles out of the water, however you see fit. You can arch your back a little bit, or you can put some ice on your balls in a flannel or use a proper ice pack.

MAHA agenda scores major win with announcement from food giant



Robert F. Kennedy Jr.'s efforts to Make America Healthy Again have consistently been paying off in the steep uphill battle against Big Pharma and Big Food. This week, a large food manufacturer joined the ranks of corporations making big changes to their ingredients in the furtherance of the MAHA agenda.

On Monday, Tyson Foods announced that it will be removing some junk ingredients from several of its brands.

'Our decision to remove high fructose corn syrup and other ingredients reflects our ongoing commitment to feeding the world like family.'

The press release states that it plans to remove high fructose corn syrup, sucralose, butylated hydroxyanisole/butylated hydroxytoluene, and titanium oxide by the end of 2025.

“We continuously review and assess our product portfolio to ensure the highest quality products that meet the needs of consumers,” said Donnie King, president and CEO of Tyson Foods. “Our decision to remove high fructose corn syrup and other ingredients reflects our ongoing commitment to feeding the world like family, while preserving the taste, value, and integrity that define our iconic brands.”

RELATED: It's been a year since Kennedy and Trump joined forces. Here are MAHA's top 3 wins.

Photo by Andrew Harnik/Getty Images

While the press release insists that all of these ingredients are FDA-approved and safe to use, critics have raised concerns over their safety in higher doses and extended periods of time. In particular, scientists have questioned the toxicity levels of BHA and BHT.

Titanium dioxide, according to the Industrial Plating Company's website, "is a hard and dense ceramic coating. It is used for a variety of coating applications, including wear resistance and electrically conductive coatings." When it is not being used for industrial-grade ceramic coatings, food companies have used it as an additive to increase the whiteness or opacity of food.

Among the brands that will see changes to their ingredients are Tyson, Jimmy Dean, Hillshire Farm, Wright, State Fair, Aidells, and ibp, according to the press release.

Earlier this year, Tyson Foods announced that it would remove "petroleum-based synthetic dyes from its domestic-branded products."

Tyson Foods did not immediately respond to Blaze News' request for comment.

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